The Precise Mechanism of the Penis Enlargement Response - PEGym

by Dr. Richard R. Howard II

Dr. Howard is the leading penis enlargement coach at YourPenisDoctor.com, providing consultations daily to his clients. In addition, Dr. Howard has been a health and fitness adviser for 10 years, at the Howard Medical Clinic, and has personally gained over 3 inches in length and 2 inches in girth.

To understand the precise mechanism of the penis enlargement response, it?s important to first understand the anatomy of the penis. The penis is a specialized vascular framework constructed to inflate to a rigid state called ? erection. The shaft consists of three chambers:

• The two chambers of the?corpus spongiosum (CS) and
• The single chamber of the corpus cavernosum (CC).

Within these chambers are prominent slit like spaces, rich in blood cells, as they are really modified veins called vascular plexus. The slit like vascular spaces are composed of an abundant amount of smooth muscle with lesser amounts of fibrous tissue, lined by endothelium. This is called erectile tissue.

Separating the CC and CS is a very thick band of connective tissue called the tunica albuginea. This connective tissue is composed mostly of bi-layered collagen and elastic fibers. The elastic fibers form an irregularly latticed network on which collagen fibers rest. It is this elastic network that gives the penis elasticity, allowing it to expand and contract, and is an important factor in the penis enlargement response.

The limiting factor of the penis enlargement response is that the composition of the tunica albuginea and septum is collagen and elastic tissue. The collagen is very tough connective tissue, constructed by nature, to ensure optimum structural integrity and strength.? Think of a tendon or ligament. They?re also composed of collagen. Your body?s entire structural framework is composed mostly of collagen, including bone.

To facilitate the penis enlargement response, you must overcome the structural bonds of the collagen.? Through sufficient traction, called critical traction, applied consistently along the length and diameter of the penis, the molecular bonds of the collagen will separate and undergo remodeling to a larger size.

The interior tissue of the penis is composed mainly of the vascular plexus, consisting of epithelium lining, with abundant smooth muscles and some fibrous tissue. When sufficient traction is applied to the collagen, this results in a controlled bond breaking and separation. The penis enlargement response and the remodeling process begins, making the collagen longer and larger.

The penis enlargement response is like taking apart a brick wall and adding another layer of bricks to increase its size.

Think of separating a brick wall and filling it in with additional bricks resulting in a larger brick wall. During the penis enlargement response, the interior vascular plexus goes along for the ride, but itself is transformed into greater mass.

The endothelium lining of the vascular slits undergoes mitosis, during the penis enlargement response. The abundant smooth muscle framework adjacent to the endothelium undergoes hypertrophy and mitosis (there are studies that support the proposition that mitosis can occur in smooth muscle cells), and the arteries and veins follow the same growth course mentioned above, with? the remaining fibrous tissue undergoing remodeling.

The slow-growing, peripheral nerves grow and elongate when stimulated during the penis enlargement response. Therefore, the entire package gets bigger under sufficient constructive, critical traction, with the collagenous structure being the limiting factor.? If the collagen bond breaking is sufficient, with cementing, the process is likely permanent.

Let?s take a brief look at ?newbie gains? and the penis enlargement response.

A ?newbie? is a person who has never tried penis enlargement. Once he begins, he will experience characteristically easy gains. Unchallenged or unstressed collagen has slack or crimps, allowing a stretching out scenario that facilitates the penis enlargement response.When these crimps are removed, it results in quick gains for the beginner. Under this condition, the collagen bonds generally remain intact until the critical traction level is applied.

Another factor in the penis enlargement response is the ligaments. The attachment points for the penis are composed of collagenous tissue, called ligaments, and skeletal muscular tissue. The?main ligaments are called ?suspensory ligaments and fundiform ligaments. The primary base muscles are called? the bulbocavernosus and ischiocavernosus muscles.

Ligaments are composed of densely-packed collagen fibers, which are white and relatively inelastic. Mechanical properties vary with shape and structural organization.?This connective tissues is characterized by sparse cellularity, and distributed within an extracellular matrix.

Cells in tendons and ligaments are called fibroblasts. These ligaments consist of 70-80% collagen and 20% fibroblasts. Upon initiating traction, the crimps of the ligament collagen are pulled out or straightened first, as part of the penis enlargement response. After this, additional traction triggers molecular transformation via molecular bond breaking and remodeling, again resulting in longer, larger ligaments, as the penis enlargement response.

Arm curls building your triceps and biceps are similar to the effect of traction and the penis enlargement response.

Another factor in the penis enlargement response is the skeletal muscles. The skeletal muscles called the bulbocavernosus and ischiocavernosus muscles, are attachment points at the base of the penis. For this reason, traction will elicit the same changes in the penis?s skeletal muscles as arm curls will elicit in your biceps and triceps. Whilst under traction, all muscular tissue at the base of the penis will undergo hypertrophy, strengthen?and thus initiate the penis enlargement response.

References

1. Frank H. Netter, M.D., The Netter Collection of Medical Illustrations,? Reproductive System, 1997; Volume 2, Page 9.
2. Gerald Brock, Geng-Long Hsu, Lora Nunes, Burkhard von Heyden, Tom F. Lue -?The Anatomy of the Tunica Albuginea in the Normal Penis and Peyronie?s Disease? -? J Urol. 1997; Volume 157, Issue 1, 276-281
3. Robert F. Diegelmann, PhD, Collagen Metabolism, ?Medical College of Virginia, Virginia Commonwealth University, Richmond, VirginiaPosted: 02/04/2002; Wounds.?2001;13(5)???2001?Health Management Publications, Inc.
4. Tetsuya Nakatani Takashi Marui Toshiaki Hitora Minoru Doita Kotaro Nishida Masahiro Kurosaka. Mechanical stretching force promotes collagen synthesis by cultured cells from human ligamentum flavum via transforming growth factor??1. J? Orthopaedic Res. November 2002; Volume 20, Issue 6, Pages: 1380?1386.
5. Hsu?G-L , Brock?GB , Martinez-Pineiro?L , Nunes?L , von Heyden?B , Lue?TF . The three-dimensional structure of the tunica albuginea: anatomical and ultrastructural levels. . Int. J. Impotence Res. . 1992;4:117 .
6. Balestrini JL,? Billiar KL. Magnitude and duration of stretch modulate fibroblast remodeling.?? J Biomech Eng. 2009 May;131(5):051005.
7. Baek SR et al, J Elast 80:13?31, 2005.
8. Driessen NJB et al, Biomechan Model Mechanobiol.7:93?103, .2008.
9. ?Humphrey JD et al, Math Models Methods Appl Sci 12:407?430, 2002

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Source: http://www.pegym.com/articles/precise-mechanism-pe-response-manual-mechanical-intervention

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